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Fig. 4 Bryostatin- 1-induced p-PKCδT505, p-PKDS744/748, <t>p-PKDS916,</t> and p-eNOSS1177 in HAECs is attenuated by rottlerin. HAECS were treated with vehicle (Veh, DMSO + ethanol), 50 nM bryosta- tin-1 (Bry), or Bry + 10 μM Rottlerin (Bry + Rot). Representative images (A) and mean densitometry (B- D) are shown. Bry-induced increases in p-PKCδT505, p-PKDS744/748, p-PKDS916, and p-eNOSS1177 were attenuated by concurrent treatment with Rot. For B-E, *p < 0.05, **p < 0.01. p values were obtained using a one-way ANOVA, followed by a Tukey post- hoc test when appropri- ate. Symbols within each histogram represent one 100 mm dish.
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Fig. 4 Bryostatin- 1-induced p-PKCδT505, p-PKDS744/748, <t>p-PKDS916,</t> and p-eNOSS1177 in HAECs is attenuated by rottlerin. HAECS were treated with vehicle (Veh, DMSO + ethanol), 50 nM bryosta- tin-1 (Bry), or Bry + 10 μM Rottlerin (Bry + Rot). Representative images (A) and mean densitometry (B- D) are shown. Bry-induced increases in p-PKCδT505, p-PKDS744/748, p-PKDS916, and p-eNOSS1177 were attenuated by concurrent treatment with Rot. For B-E, *p < 0.05, **p < 0.01. p values were obtained using a one-way ANOVA, followed by a Tukey post- hoc test when appropri- ate. Symbols within each histogram represent one 100 mm dish.
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Fig. 4 Bryostatin- 1-induced p-PKCδT505, p-PKDS744/748, <t>p-PKDS916,</t> and p-eNOSS1177 in HAECs is attenuated by rottlerin. HAECS were treated with vehicle (Veh, DMSO + ethanol), 50 nM bryosta- tin-1 (Bry), or Bry + 10 μM Rottlerin (Bry + Rot). Representative images (A) and mean densitometry (B- D) are shown. Bry-induced increases in p-PKCδT505, p-PKDS744/748, p-PKDS916, and p-eNOSS1177 were attenuated by concurrent treatment with Rot. For B-E, *p < 0.05, **p < 0.01. p values were obtained using a one-way ANOVA, followed by a Tukey post- hoc test when appropri- ate. Symbols within each histogram represent one 100 mm dish.
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Fig. 4 Bryostatin- 1-induced p-PKCδT505, p-PKDS744/748, <t>p-PKDS916,</t> and p-eNOSS1177 in HAECs is attenuated by rottlerin. HAECS were treated with vehicle (Veh, DMSO + ethanol), 50 nM bryosta- tin-1 (Bry), or Bry + 10 μM Rottlerin (Bry + Rot). Representative images (A) and mean densitometry (B- D) are shown. Bry-induced increases in p-PKCδT505, p-PKDS744/748, p-PKDS916, and p-eNOSS1177 were attenuated by concurrent treatment with Rot. For B-E, *p < 0.05, **p < 0.01. p values were obtained using a one-way ANOVA, followed by a Tukey post- hoc test when appropri- ate. Symbols within each histogram represent one 100 mm dish.
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Image Search Results


Journal: Pathogens

Article Title: Antimicrobial Potential of Bee-Derived Products: Insights into Honey, Propolis and Bee Venom

doi: 10.3390/pathogens14080780

Figure Lengend Snippet: Selection of articles on the antimicrobial effect of honey.

Article Snippet: 37 Polish monofloral honeys , S. aureus PCM 2051, S. epidermidis PCM 2118, P. aeruginosa ATCC 27853, E. coli K12 , In vitro-broth microdilution assay, , Out of the tested honey samples, cornflower, thyme, buckwheat and tansy phacelia honeys exhibited the strongest inhibitory effect, especially against staphylococci. The antibacterial effect was strongly correlated to honey color and phenolic content. Some samples presented greater antibacterial potency than manuka honey. Antibacterial activity was determined to be primarily hydrogen peroxide dependent. , [ ] .

Techniques: Selection, Dilution Assay, Inhibition, Diffusion-based Assay, Activity Assay, Formulation, Microdilution Assay, Concentration Assay, Control, Comparison, In Vivo, High Performance Thin Layer Chromatography

Fig. 4 Bryostatin- 1-induced p-PKCδT505, p-PKDS744/748, p-PKDS916, and p-eNOSS1177 in HAECs is attenuated by rottlerin. HAECS were treated with vehicle (Veh, DMSO + ethanol), 50 nM bryosta- tin-1 (Bry), or Bry + 10 μM Rottlerin (Bry + Rot). Representative images (A) and mean densitometry (B- D) are shown. Bry-induced increases in p-PKCδT505, p-PKDS744/748, p-PKDS916, and p-eNOSS1177 were attenuated by concurrent treatment with Rot. For B-E, *p < 0.05, **p < 0.01. p values were obtained using a one-way ANOVA, followed by a Tukey post- hoc test when appropri- ate. Symbols within each histogram represent one 100 mm dish.

Journal: GeroScience

Article Title: Bryostatin-1 improves function in arteries with suppressed endothelial cell autophagy.

doi: 10.1007/s11357-025-01650-5

Figure Lengend Snippet: Fig. 4 Bryostatin- 1-induced p-PKCδT505, p-PKDS744/748, p-PKDS916, and p-eNOSS1177 in HAECs is attenuated by rottlerin. HAECS were treated with vehicle (Veh, DMSO + ethanol), 50 nM bryosta- tin-1 (Bry), or Bry + 10 μM Rottlerin (Bry + Rot). Representative images (A) and mean densitometry (B- D) are shown. Bry-induced increases in p-PKCδT505, p-PKDS744/748, p-PKDS916, and p-eNOSS1177 were attenuated by concurrent treatment with Rot. For B-E, *p < 0.05, **p < 0.01. p values were obtained using a one-way ANOVA, followed by a Tukey post- hoc test when appropri- ate. Symbols within each histogram represent one 100 mm dish.

Article Snippet: The membrane was blocked using 5% BSA for 1 h at room temperature and incubated with: (i) primary antibodies to LC3 (L8918, Sigma-Aldrich, St. Louis, MO, USA), p62 (ab91526, Abcam, Waltham, MA, USA), p-eNOSS1177 (#MA5-14957, ThermoFisher, Waltham, MA, USA), eNOS (#9572, Cell Signaling, Boston, MA, USA), GAPDH (#2118, Cell Signaling, Boston, MA, USA, p-PKDS744/748 (#2054, Cell Signaling, Boston, MA, USA), p-PKDS916 (#2051, Cell Signaling, Boston, MA, USA), and p-PKCδT505 (#9374, Cell Signaling, Boston, MA, USA); followed by (ii) secondary antibodies to Goat anti-Rabbit and Goat anti-Mouse (for experiments involving PKD inhibition).

Techniques: